Saturated fat

5 easy clues that can help you decide how much fat you need to eat

When I speak on saturated fat, I usually get plenty of questions. As I have said, “A high saturated fat diet kills trillions of good microbes in the gut (specifically, Bifidus) and allows a pathogenic bacterium named B. wadsworthia to flare up, produce toxic substances (LPS), and inflame the gut lining, even causing leaky gut.”

GI Distress Relief

Eating too many saturated fats from animals may destroy the diversity of your microbiome. But hardy Bifidus bacteria, like those found in our GI Distress Relief probiotic, can help protect the gut from inflammation caused by excess fat in the diet.

During the hot summer months, you need little-to-no saturated fat from animals, and all your fat should come from plants.

Let’s look at the research to clear up confusion

A 2012 study published in Nature confirms this. When researchers took mice with a genetic predisposition similar to colitis and fed them a diet high in saturated fat, the bacterial composition of the microbiome changed and encouraged the growth of a harmful bacteria called B. wadsworthia.1

More B. wadsworthia in the gut, researchers said, can trigger an inflammatory immune response in mice predisposed to the disease. The fat used in this study was milk fat, found in butter, cream, ice cream, cheese, and high-fat milk.

A more recent American Journal of Clinical Nutrition study, published in 2020, also indicated that even one high-saturated-fat meal — with fat content similar to a fast food meal — could be enough to affect concentration.2

Whether saturated fat is good for you or not is a question on the minds of many today, so let’s dig deeper.

In the last few years, fat has gone from demonized to the darling of many health experts. You’ve probably been hearing these statements everywhere you turn:

  • A diet that provides energy from carbs is bad.
  • A high-fat ketogenic diet is the way to go.
  • Fat should be eaten at every meal.

This entire fat issue is bewildering to many, and it’s certainly not easy to find answers amidst the conflicting opinions. So, I’d like to chime in with another piece of the fat story and hopefully put an end to the debate.

Are you clear about where fats come from and why we eat them?

There are different types of fat — saturated, unsaturated, monounsaturated, polyunsaturated, and even more than you probably want to remember — so let’s forget about that right now. No wonder it’s confusing.

We eat fats:

  • As a source of energy.
  • Because they help keep our cell membranes more flexible.
  • Because they supply essential fatty acids — since our bodies can’t make the essential fatty acids like omega-3 and omega-6 on their own.
  • For cholesterol — to form steroid hormones, like progesterone, estrogens, and testosterone.
  • For the proper functioning of our nerves and brains.
  • To carry fat-soluble vitamins, like vitamins A, D, E, and K, around the bloodstream, delivering them to the cells.
  • To store as energy for later use.

Remembering that fats are a very important part of a healthy diet and remembering a few more facts is wise. Such as:

  • Fats can be obtained from both plants and animals.
  • Saturated fat can be obtained from just two plants but from many animals.
  • The healthiest saturated fats are in fatty fish, like salmon, and in plant sources, like coconut oil, coconut milk, and MCT oil. Red palm oil is also a saturated fat, and while it isn’t as popular as coconut and MCT oil are right now, it’s still good to eat. Research shows that heart health is most likely negatively impacted by the saturated fats found in meat and not in plants.3,4
  • Three fats that are excellent for our health that are not saturated come from these plants: extra virgin olive oil; avocado oil, which is extracted from the flesh of the avocado, not the pit; and macadamia nut oil, which has a delicious and buttery flavor. These fats are monounsaturated (MUFAs). It’s these that you’ll want to eat more of if you choose to eat a high-fat diet.

When you hear experts like Dr. Mark Hyman promoting a high-fat diet, these are the fats he is recommending, not saturated fat from commerciallyraised animals. That kind of fat is bad for everyone.

Yet if those animals are pasturefed on green grasses most of their life, they will provide you with a healthier type of saturated fat, plus some omega-3 fatty acids, and this could be good for you.

Notice I used the word “could.” That’s because it may or may not be good for you. And this is where your own individual uniqueness comes in (a concept I’ve tried to promote for years in The Body Ecology Principle of Uniqueness).

Download our Blueprint for more information on what Body Ecology is all about.

So, how much fat should you eat? 5 simple ways to tell

Along with your uniqueness, consider:

1. Climate.

Both where you live and the season of the year can be factors to consider, especially when it comes to consuming saturated fats:

  • The winter is a time to take in more saturated fat from animals since doing so will help you stay warm during the cold months; cool temperatures may alter metabolism and increase brown fat storage.5
  • During the hot summer months, you need little-to-no saturated fat from animals, and you’ll feel better if your fat comes from plants.

This same concept — of changing the type of fat you eat — holds true if you live in a hot climate most of the time, or if you live where seasons come and go. If and when it’s cold in your area, saturated fat from animals can help you stay warmer.

Bottom line: If you live in Hawaii, ditch the saturated fat from animals.

Don’t take my word for it — try it. During hot weather, eat a quinoa salad one night and then a beef stew the next and see how much better you feel and sleep. Also, notice with plant fats that you’ll be much more tolerant of the summer heat.

Remember, coconuts grow in hot, tropical climates. For people living in hotter climates, this is a strong clue from Mother Nature that your saturated fat should come from coconuts and not from animals.

In the summertime, try this millet tabouli salad with an olive oil or macadamia nut oil dressing. Quinoa makes a lovely summer salad too. Quinoa and millet are both Body Ecology-recommended grain-like seeds; they’re gluten-free and are not carbohydrates.

2. Exercise.

The kind and amount of exercise you do each week must be considered when you decide how much fat to eat. You’ll notice that many of the advocates of a very high-fat diet are athletes and burn a lot of fat for fuel.6 Do you?

3. Food combining.

Do you have an untamable sweet tooth and eat sugar every day? Fats combined with sugar are a big no-no. Adding sugar to an already high-fat Western diet may be even more harmful than eating a high-fat Western diet alone.7

4. Genes.

Have you had your genes tested yet? You might want to. If you’ve inherited “variants” (a.k.a. SNPs or alleles) in certain genes, then saturated fats from animals are probably not for you.

If you have any of the following gene SNPs, you’ll want to carefully monitor your fat intake:

  • APOA2 – The variant in this gene is C and is associated with weight gain when saturated fat intake is high, as well as insulin resistance and atherosclerosis.
  • APOA5 – The T variant in this gene has been associated with greater weight gain and less weight loss when eating a high-saturated fat diet.
  • APOe4 – This is the gene that puts many at risk for Alzheimer’s and cardiovascular disease.8 A low-fat diet is recommended for this genotype, which means no saturated fats from animals and little or none of the healthier fats from plants.

    Even omega-3 fats from fresh fish like salmon are a concern. So, perhaps someone with this gene should not take fish oil supplements and should obtain their omega-3s from plants like algae and algal powder.

  • FABP2 – This gene, which stands for fatty-acid-binding protein 2, strongly influences fat absorption in the small intestine. With the A variant, you will absorb more fat in your small intestine and tend to gain weight. It seems unfair, but if you have the A variant and share the same moderate-fat meal with a friend who does not have this variant, you’ll absorb more fat.

    You will naturally struggle with more weight gain and increased abdominal fat than he or she will; eating a high-fat diet will cause weight gain and abdominal fat storage for you, and even a moderate amount of fat will be better absorbed. The FABP2 gene A variant is also associated with insulin resistance and higher triglycerides when fat is eaten.9

  • FTO – This well-researched gene has been associated with a higher risk for a larger waist circumference if you have the A variant and eat saturated fat.10 With the A variant, you have to carefully manage your total fat intake.

    It’s also important to avoid sweet foods and flour products, and you must decrease the amount of saturated fats from even grass-fed animals, while including more MUFAs (monounsaturated fats). Think avocado and avocado oil, extra virgin olive oil, and macadamia nut oil.

5. Your liver, pancreas, and gut microbiome.

These three organs play a key role in your ability to digest the fats you eat:

  • In 2012, researchers learned that some microbes in the gut can increase dietary fat absorption to help the body extract more calories from the same amount of food.11
  • Supporting the gut with good bacteria, the study indicated, could be used to decrease fat absorption and reduce instances of metabolic disease.

Let’s also not forget — as most experts do — that the condition of your liver, pancreas, and the substances they produce, like bile and pancreatic enzymes, are other key players in how well you digest fats.

A keto diet beats a high-carb diet every time

If you’re on The Body Ecology Diet — as I have been for over two decades now — you are on a “modified ketogenic diet.”

You and I are obtaining our energy from our fats, not from carbs. That’s because:

  • Body Ecology is sugar-free (no carbs) and gluten-free (and The Diet doesn’t recommend any flour products either).
  • Throughout each day, those of us on The Body Ecology Diet consume 80 percent of our food from vegetables, eating many different plants (dark, green leafy; cruciferous; root vegetables, like daikon and carrots; and ocean vegetables).
  • Body Ecology has a strong emphasis on supporting gut health, and this is an area where The Body Ecology Diet shines far above the other advocates of the ketogenic diet, who don’t fully understand the value of fermented foods.
  • The healthy microbes in cultured veggies and probiotic drinks help consume sugars and break down fats and proteins so they’re more digestible.

I stand firmly on the statement I made that a diet high in saturated fat from animals (and usually eaten with carbohydrates) is going to destroy the diversity and health of your microbiome.

A high-fat diet decreases the number of Bifidus bacteria, an important member of a healthy inner ecosystem. Bifidobacteria have been shown to improve intestinal barrier function and decrease toxic substances that cause intestinal inflammation called lipopolysaccharides (LPS produced by pathogenic bacteria).12

They have also been shown to play a role in regulating high-fat-induced diabetes.12 Prebiotics, such as the Sunfiber included in our GI Distress Relief formula, can help increase the number of Bifidobacteria and reduce the impact of metabolic disorders caused by a high-fat diet.12

Following The Body Ecology Way of Living, which is a high-fiber, 80-percent plant food way of eating combined with healthy, natural fats — from avocados, avocado oil, macadamia nut oil, extra virgin olive oil, coconut milk, coconut oil, MCT oil, fatty fish, grass-fed meats, and a small amount of seeds and nuts — will help you restore and maintain your inner ecology.


  1. 1. Devkota S, Wang Y, Musch MW, Leone V, Fehlner-Peach H, Nadimpalli A, Antonopoulos DA, Jabri B, Chang EB. Dietary-fat-induced taurocholic acid promotes pathobiont expansion and colitis in Il10-/- mice. Nature. 2012 Jul 5;487(7405):104-8.
  2. 2. Janice K Kiecolt-Glaser, Michael T Bailey, William B Malarkey, Megan E Renna, M Rosie Shrout, Rebecca Andridge, Martha A Belury, Annelise A Madison. Afternoon distraction: a high-saturated-fat meal and endotoxemia impact postmeal attention in a randomized crossover trial. The American Journal of Clinical Nutrition, 2020; DOI: 10.1093/ajcn/nqaa085.
  3. 3. Jaike Praagman, Linda E.T. Vissers, Angela A. Mulligan, Anne Sofie Dam Laursen, Joline W.J. Beulens, Yvonne T. van der Schouw, Nicholas J. Wareham, Camilla Plambeck Hansen, Kay-Tee Khaw, Marianne Uhre Jakobsen, Ivonne Sluijs. Consumption of individual saturated fatty acids and the risk of myocardial infarction in a UK and a Danish cohort. International Journal of Cardiology, 2018; DOI: 10.1016/j.ijcard.2018.10.064.
  4. 4. Jun Li, Qi Sun. Consumption of saturated fatty acids and coronary heart disease risk. International Journal of Cardiology, 2019; DOI: 10.1016/j.ijcard.2019.01.022.
  5. 5. Lee P, Smith S, Linderman J, Courville AB, Brychta RJ, Dieckmann W, Werner CD, Chen KY, Celi FS. Temperature-acclimated brown adipose tissue modulates insulin sensitivity in humans. Diabetes. 2014 Nov;63(11):3686-98. doi: 10.2337/db14-0513. Epub 2014 Jun 22.
  6. 6. Jeff S. Volek, Daniel J. Freidenreich, Catherine Saenz, Laura J. Kunces, Brent C. Creighton, Jenna M. Bartley, Patrick M. Davitt, Colleen X. Munoz, Jeffrey M. Anderson, Carl M. Maresh, Elaine C. Lee, Mark D. Schuenke, Giselle Aerni, William J. Kraemer, Stephen D. Phinney. Metabolic characteristics of keto-adapted ultra-endurance runners. Metabolism, 2015; DOI: 10.1016/j.metabol.2015.10.028.
  7. 7. Raffaella Crescenzo, Francesca Bianco, Paola Coppola, Arianna Mazzoli, Margherita Tussellino, Rosa Carotenuto, Giovanna Liverini, and Susanna Iossa. Fructose supplementation worsens the deleterious effects of short term high fat feeding on hepatic steatosis and lipid metabolism in adult rats. Experimental Physiology, June 2014 DOI: 10.1113/expphysiol.2014.079632.
  8. 8. Kim J, Basak JM, Holtzman DM. The Role of Apolipoprotein E in Alzheimer’s Disease. Neuron. 2009;63(3):287-303. doi:10.1016/j.neuron.2009.06.026.
  9. 9. Weiss EP, Brown MD, Shuldiner AR, Hagberg JM. Fatty acid binding protein-2 gene variants and insulin resistance: gene and gene-environment interaction effects. Physiol Genomics. 2002 Sep 3;10(3):145-57. Review.
  10. 10. Fawcett KA, Barroso I. The genetics of obesity: FTO leads the way. Trends in Genetics. 2010;26(6):266-274. doi:10.1016/j.tig.2010.02.006.
  11. 11. Ivana Semova, Juliana D. Carten, Jesse Stombaugh, Lantz C. Mackey, Rob Knight, Steven A. Farber, John F. Rawls. Microbiota Regulate Intestinal Absorption and Metabolism of Fatty Acids in the Zebrafish. Cell Host & Microbe, 2012; 12 (3): 277 DOI: 10.1016/j.chom.2012.08.003.
  12. 12. Cani PD, Neyrinck AM, Fava F, Knauf C, Burcelin RG, Tuohy KM, Gibson GR, Delzenne NM. Selective increases of bifidobacteria in gut microflora improve high-fat-diet-induced diabetes in mice through a mechanism associated with endotoxaemia. Diabetologia. 2007 Nov;50(11):2374-83.

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